Intestinal Angioedema

Angioedema is a noninflammatory disease characterized by increased capillary permeability with extravasation of intravascular contents leading to edema. The face, limbs, and airways may be involved and angioedema of the latter can cause life threatening airway obstruction. Intestinal involvement is also reported and can present with acute abdomen, or in rare cases, hypovolemic shock.

Angioedema is due to hereditary and idiopathic, or secondary to therapy with angiotensin converting enzyme (ACE) inhibitors in patients with hypertension.

CECT findings include bowel wall and mucosal thickening, enhancement of the mucosa (differentiates from ischemic bowel wall thickening), prominent mesenteric vessels, and ascites. 


REFERENCES
De Backer AI, De Schepper AM, Vandevenne JE, et al. CT of angioedema of the small bowel. AJR Am J Roentgenol 2001;176(3):649-52.
Scheirey CD, Scholz FJ, Shortsleeve MJ, et al. Angiotensin-converting enzyme inhibitor-induced small-bowel angioedema: clinical and imaging findings in 20 patients. AJR Am J Roentgenol 2011;197(2):393-8.

Isodense Subdural Hematoma

The above NECT images demonstrate a right sided extra-axial crescentic collection that has similar attenuation to gray matter. The collection causes effacement of right sided sulci, and mass effect with midline shift. The isodensity to underlying cortical tissue makes this consistent with an isodense/subacute subdural hematoma indicating that its age ranges from ~3 days to 3 weeks. In patients with anemia, an isodense subdural hematoma can occur in the acute setting.


REFERENCES
Kim KS, Hemmati M, Weinberg PE. Computed tomography in isodense subdural hematoma. Radiology 1978;128:71-4.

Causes of Hepatic Capsular Retraction

Peripheral Cholangiocarcinoma

• malignant tumor arising from interlobular bile ducts
• peripheral enhancement on arterial phase CECT with gradual centripetal fill in
• delayed enhancement is common due to fibrotic regions within mass
• may have dilated bile ducts upstream from the lesion

Confluent Hepatic Fibrosis

• occurs in the setting of cirrhosis
• usually in the medial segment of the left lobe and/or anterior segment of the right lobe
• peripheral wedge shaped regions of low attenuation on CECT that show delayed enhancement
• biliary ductal dilatation not seen

Metastases

• treated adenocarcinoma (usually colorectal, breast) can cause hepatic capsular retraction 
• regions of fibrosis (pseudo-cirrhosis) may also be seen

REFERENCES

Fennessy FM, Moretele KJ, Kluckert T, et al. Hepatic capsular retraction in metastatic carcinoma of the breast occurring with increase or decrease in size of subjacent metastasis. AJR Am J Roentgenol 2004;182(3):651-5.

Lee JW, Kim S, Kwack SW, et al. Hepatic capsular and subcapsular pathologic conditions: demonstration with CT and MR imaging. Radiographics 2008;28:1307-23.

Lipson JA, Qayyum A, Avrin DE, et al. CT and MRI of hepatic contour abnormalities. AJR Am J Roentgenol 2005;184(1):75-81.

Achalasia

Achalasia is a disorder of esophageal dysmotility due to failure of the gastroesophageal sphincter to relax because of destruction of its myenteric plexus. Primary achalasia is considered idiopathic while secondary achalasia could be due to destruction of the myenteric plexus by tumor or infection (Chagas disease). The radiologic diagnosis of primary achalasia is best made with barium studies which will show marked dilation of the esophagus with smooth narrowing near the GE junction (bird's beak deformity). Real time imaging will also show absent primary peristalsis and the presence of tertiary contractions. While cross sectional imaging is not the most sensitive in diagnosing achalasia, sometimes the finding is made incidentally as in the above case. The differential diagnosis of achalasia was previously discussed here.


REFERENCES
Noh MH, Fishman EK, Forastiere AA, et al. CT of the esophagus: spectrum of disease with emphasis on esophageal carcinoma. Radiographics 1995;15:1113-34.
Woodfield CA, Levine MS, Rubesin SE, et al. Diagnosis of primary versus secondary achalasia: reassessment of clinical and radiographic criteria. AJR Am J Roentgenol 2000;175(3):727-31.

Killian Jamieson Diverticulum

Killian is usually mentioned when discussing the Killian's dehiscence which is the gap in the posterior hypopharynx where the muscle fibers of the cricopharyngeus muscle and inferior constrictor muscle diverge. This site is where Zenker's diverticula occur.

The Killian-Jamieson space is below the cricopharyngeus muscle and lateral to the longitudinal muscle of the esophagus. This muscular gap is the location of the rarer Killian-Jamieson diverticulum. Fluoroscopy will show an outpouching along the anterolateral esophagus inferior to the cricopharyngeus muscle (as opposed to along the posterior esophagus and superior to the cricopharyngeus muscle in the case of a Zenker's diverticulum). 

REFERENCES

Rubesin SE and Levine MS. Killian-Jamieson diverticula. AJR Am J Roentgenol 2001;177:85-9.

SATCHMO

The differential diagnosis of a suprasellar mass can be remembered through the SATCHMO mnemonic.

S - suprasellar/sellar adenoma, sarcoid
A - aneurysm, arachnoid cyst
T - teratoma
C - craniopharyngioma
H - hamartoma (of the tuber cinereum), hypothalamic glioma
M - meningioma
O - optic nerve glioma

The above image is from a patient who presented to the ER several times after multiple motor vehicle accidents. On clinical exam he was found to have visual field defects. NECT demonstrates a hyperattenuating sellar mass. Post operative diagnosis was pituitary macroadenoma.

Gossypiboma

Stemming from the Latin gossypium, meaning cotton, gossypiboma refers to retained cotton products such as surgical sponges. A gossypiboma can elicit an inflammatory response leading to formation of an intraabdominal abscess, or cause obstructive symptoms due to mass effect.


REFERENCES
Manzella A, Filho PB, Albuquerque E, et al. Imaging of gossypibomas: pictorial review. AJR Am J Roentgenol 2009;193:S94-101.